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Furthermore, in certain tissues, like muscles, mTORC1 activity even declines with age, in part due to the development of anabolic resistancea major cause for muscle wasting in the elderlywith mTORC1 requiring higher AA amounts to maintain proper cellular metabolic activities (Dardevet et al., 2012)

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Regardless, our finding that expression of mitochondrially-targeted CHAC1 is sufficient to restore ferroptosis sensitivity in CHAC1-deficient cells indicates that the maintenance of matrix cysteine availability is operative in the induction of ferroptosis

Similarly, in a spared nerve injury (SNI)-induced NP rat model, reduced GPX4 expression contributed to the onset of ferroptosis

Yi Huang, [email protected] This article was submitted to Epigenomics and Epigenetics, a section of the journal Frontiers in Genetics Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

The single intranigral injection of LPS as a new model for studying the selective effects of inflammatory reactions on dopaminergic system