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We speculate that this specific residue, which is located in the linker region of human parkins sequence, could play a key role in its folding, in interactions with other molecules, and thus in its functions, akin to the phosphorylation at serine 65 during toxin-induced mitophagy in mammalian cells [3, 30, 34]

Streu, C

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SLU-PP-332 is evaluated in laboratory research to study ERR receptor and energy expenditure pathway research

And what get a little bit worse and what you see in the literature is that as people age, starting around age 18 through age 70 to 80, youre losing your mitochondrial function an average rate of 1% per year