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NAC protected against oxidative stress through acting on this newly disclosed Nrf2/GR/GSH pathway, by which NAC elevated the biosynthesis of protective GSH to repair and reconstitute the defense system destroyed by phosgene

This vicious cycle creates a metabolic hyperuricemia phenotype, characterized by stimulated hepatic lipogenesis and dysregulated adipokine levels

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On the other hand, the HIF-1/HIF-1 dimer, together with the signal transducer and activator of transcription 3 (STAT3), an inducer of HIF-1 stability, participate in the genetic transcription of VEGF by binding to its activator CBP/p300