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In this regard, our laboratory has identified deep, adenomyotic lesions in our mouse model of developmental toxicant exposure also presenting with an endometriosis-like uterine phenotype (Nayyar et al., 2007

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Stadtman ER, Berlett BS

R., Ritter, H

The same logic applies here

In conclusion, future research efforts should focus on unraveling the complex interactions between oxidative stress and other key pathways in TAO, identifying new therapeutic targets, and conducting robust clinical studies to validate the effectiveness of antioxidant interventions