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Upon entering the cytoplasm, the chloride atoms on the drug are displaced by water molecule, resulting in a compound that binds to the N7 reactive center on purines and causes DNA damage

doi: 10.1186/s12885-016-2228-y 73 FontanaFEsserAKEgbulefuCKarmakarPSuXAllenJSet al

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Year : 2016 Computational Modeling of Single Neuron Extracellular Electric Potentials and Network Local Field Potentials using LFPsim Cite this Research Publication : Harilal Parasuram, Bipin G

At this point, we have previously shown that IGF-I is a main character in restoring mitochondrial dysfunction during aging by increasing mitochondrial membrane potential, reducing oxygen consumption, and increasing ATP synthesis what in turn minimize the c-cytochrome release to the cytoplasm and subsequently promote neural survival by decreasing caspase-induced apoptosis[55, 56], in agreement with in vitro reported works from others[242244]

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