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SLC7A11 disruption in PDAC cell lines strongly affects their amino acid and redox balance, and thus suppresses in vitro and delays in vivo their proliferative phenotype ( in vivo SLC7A11 knock out PDAC cells grew normally

Ezt a regenerld L-glutation semlegesti

This apparently is a host defense mechanism to prevent the suicide of the cell by the S U t U ib / . - s - also the host cell

Data availability statement The data that support the findings of this study are available on request from the corresponding author

The failure of these mitochondrial complexes in brain tissue is also expected to increase ROS production leading to further localized cellular damage

Once absorbed, it binds to plasma proteins before it is transported around the body