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The regulated eNOS-NO pathway protects ischemia-reperfusion liver injury via activating eNOS, thus increasing NO, which protects hepatocytes from the insults ( via endothelial-derived NO production ( in vivo showed that the blood flow and vascular richness of the liver margin were significantly decreased

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Future research should focus on delving deeper into the pathological mechanism of HAPH, developing more effective diagnosis and treatment methods, and ultimately providing more hope and well-being for patients

Maroof A, English NR, Bedford PA, Gabrilovich DI, Knight SC

Mocco J, Mack WJ, Ducruet AF et al (2006) Complement component C3 mediates inflammatory injury following focal cerebral ischemia

[DOI] [PMC free article] [PubMed] [Google Scholar] 28.El-Gindy Y., Zeweil H., Zahran S., El-Rahman M.A., Eisa F