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Integrative perspective on autophagy regulation Although the transcriptional and epigenetic regulation of autophagy involves a diverse array of factors, ranging from nutrient-sensing kinases and stress-responsive transcription factors to chromatin remodelers and metabolic regulators, these seemingly disparate elements converge upon several core signaling hubs and transcriptional circuits

Aspirin-treated cells are incapable of cell division, but still retain some level of metabolic activity (Alem and Douglas, 2015)

Based on these clues, we hypothesized that two broad mechanisms could potentially account for these findings: 1) intracellular acetate production from other nutrients

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