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Additionally, PTMs play vital roles in DNA mismatch repair, metabolic pathways, and immune responses in tumor cells [18]

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Venlafaxine's norepinephrine-mediated BP elevation operates through a different mechanism (sympathetic outflow), and the net hemodynamic result of combining an NO modulator with a NET inhibitor has never been studied

KRAS is an oncogenic protein that is commonly activated in many tumors, including lung cancer and pancreatic cancer, and it has been considered an undruggable target because it lacks a classical drug binding site 143,144

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