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TABLE 3 Summary of renoprotective approaches targeting autophagy/mitophagy in CP-induced AKI

These pathways were selected because they correspond to the core biological foundations of neurodegenerationproteinopathy, synaptic disfunction, neuroinflammation, mitochondrial impairment, vascular and metabolic dysregulation, among othersthereby anchoring the workflow in mechanisms that are central to dementias and making it applicable across AD, FTD, DLB, vascular and mixed dementias, as well as related neurodegenerative phenotypes that share these pathogenic mechanisms

Thus, we examined the expression of SLC7A11 at plasma membrane (PM) in VRK2 knockdown cells

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Figure 3: Some enrolling patient in our study that shown swelling joint