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CD38 dictates age-related NAD decline and mitochondrial dysfunction through an SIRT3-dependent mechanism

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Without effective Phase 3 clearance, even well-functioning Phase 1 and Phase 2 pathways can result in toxin recirculation and symptom exacerbation

The proposed theory also states that dextrose may down regulate TRPV1 receptors

36 The induction of MPTP will eventually cause the release of multiple important mitochondrial proteins into the cytoplasm, such as apoptosis-inducing factor (AIF), endonuclease G (Endo G), and cytochrome c (Cyt C)