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Wang H, Ridgway Z, Cao P, Ruzsicska B, Raleigh DP

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Removal of -amyloid from CSF can delay the progression of disease and enhance the brain functions since A deposits activates mitochondrial dysfunction pathways, and release ROS, to induce an apoptotic cascade 49,50,51

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As another important regulator of ferroptosis, the overexpression of p53 in FLSs was thought to be associated with increased invasiveness and bone destruction (69), but no studies linked p53 expression to ferroptosis in RA yet