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[DOI] [PMC free article] [PubMed] [Google Scholar] 45.Uddin MS, Mamun AA, Alghamdi BS, Tewari D, Jeandet P, Sarwar MS, et al

[DOI] [PMC free article] [PubMed] [Google Scholar] 27.Jung E.C., Kim K.D., Bae C.H., Kim J.C., Kim D.K., Kim H.H

Neurodegeneration was also found to involve this mechanism, in which astrocytes with impaired glutamate uptake possessed lower EAAT2 and GLAST expression (Hefendehl et al., 2016)

In response to neuronal injury, the TIR domain of SARM1 cleaves NAD+ to generate ADP ribose (ADPR) and cyclic ADPR, which may contribute to axonal degeneration ( In summary, there are many ways of restoring NAD+ level depletion caused by aging or other diseases, including improving NAMPT expression, providing NAD+ precursors, or inhibiting NAD+, consuming enzymatic activities of PARP, CD38, and SARM1

Astilbin Alleviates Radiation-Induced Pulmonary Fibrosis via circPRKCE Targeting the TGF-/Smad7 Pathway to Inhibit Epithelial-Mesenchymal Transition