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By restoring mitochondrial respiration, reducing reactive oxygen species and stabilizing mitochondrial membrane potential, transferred mitochondria prevent apoptosis and support survival under cytotoxic conditions [359]

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Realizing this potential requires aligning concentration with cell-type needs (25 mM for memory T cells, 10 mM for Th1 polarization), addressing confounders, and advancing targeted deliverytranslating its context-dependent mechanisms to clinical benefit

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For example, if a cellular scaffold protein is required to facilitate hydroxylation of a target protein, this will likely not be identified as a target in these assays

Performance of a multi-disciplinary emergency department observation protocol for acetaminophen overdose