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Therapeutic interventions aimed at modulating lipid availability, such as ACAT inhibitors, PPAR antagonists, or scavenger-receptor blockade, can reduce the supply of anti-ferroptotic lipid mediators and promote oxidative lipid stress within tumor cells

Additionally, NF-B and JAK/STAT signaling pathways play a central role in the pathological process of UC by regulating the expression of inflammatory factors

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The 1.5 crystal structure of human receptor for advanced glycation endproducts (RAGE) ectodomains reveals unique features determining ligand binding

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In doing so, collagen prevents the potential for metastasis