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Moreover, there is increasing evidence that ferroptosis is an autophagy-dependent form of cell death and that either intracellular transition activation or abnormally elevated lysosomal activity can cause the accumulation of intracellular iron ions and lipid peroxides, which in turn promotes ferroptosis

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This cascade subsequently activates caspase-9, followed by caspase-3 activation, ultimately resulting in neuronal apoptosis (176) (177)

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