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Our collective findings provide a greater understanding into the mechanisms that govern oxidative inactivation of GLRX and may have implications for disease conditions in which PSSG and/or GLRX status are dysregulated

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For example, as in epilepsy, neuroinflammation is an important mechanism of BBB disruption in AD patients

From a mechanistic perspective, iodine status creates a U -shaped risk curve for neurological outcomes: insufficient intake undermines hormone synthesis and cognitive capacity, while excess fosters autoimmunity and inflammatory cascades (145)

The use of angiotensin-converting enzyme (ACE) inhibitors also mandates caution with ozone therapy [20, 21]