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Disclosures: Liam Swain: Nothing to Disclose, Abdel Shaheen: Nothing to Disclose, Meng Wang: Nothing to Disclose, Jennifer Flemming: Nothing to Disclose, Mayur Brahmania: Nothing to Disclose, Juan Abraldes: Gilead: Grant/Research Support, 89bio: Consultant, Boston Pharmaceuticals: Consultant, Novo Nordisk: Consultant, Boehringer Ingelheim: Consultant, Astra Zeneca: Consultant, Agomab: Consultant, Terumo: Speaking and Teaching, Cook: Grant/Research Support, Karen Tang: Nothing to Disclose, Jenny Godley: Nothing to Disclose 1550 ENGINEERED HUMAN LIVER CO-CULTURES TO MODEL HEPATIC DYSFUNCTIONS IN METALD Owen Lally 1 , 1 University of Illinois Chicago Background: Metabolic dysfunction-associated liver disease (MASLD) with increased alcohol intake is characterized by symptoms of both MASLD and alcohol-associated liver disease (ALD), known as MetALD

All handling procedures strictly followed institutional animal ethical guidelines

Small-molecule fluorescent probes for binding-and activity-based sensing of redox-active biological metals

Fatigue reduces exercise, which leads to more muscle loss and weight gain

A low fiber diet, or low residue diet, limits the amount of fiber you eat each day

Nrf2-mediated expression of the transcription factor AKR1C1 reduced ferroptosis-induced cell injury and attenuated inflammatory responses (304)