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Collectively, this dual metabolic identity, preserving physiological specialization in citrate production while hijacking polyamine and citrate pathways for oncogenic rewiring, not only distinguishes PCa from other malignancies but also unveils targetable vulnerabilities for diagnostic and therapeutic innovation

doi: 10.3390/antiox11112167 279

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Cells were used for analysis 72 hours after transfection

3.6 Friedreichs ataxia Friedreichs ataxia (FRDA) is a progressive neurodegenerative disease characterized by oxidative stress and mitochondrial dysfunction with decreased expression of the mitochondrial protein frataxin (FXN)