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Investigating the roles of oxidative stress, inflammation and genetic susceptibility contributes to a deeper understanding of NIHL pathophysiology, potentially leading to the development of pharmaceutical interventions, gene therapies, or innovative hearing protection devices

Oxeiptosis: a novel pathway of melanocytes death in response to oxidative stress in vitiligo

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Preclinical research has also observed downregulation of pro-inflammatory cytokines including TNF-, IL-6, and IL-1, alongside modulation of NF-B signalling pathways positioning GHK-Cu as a subject of interest in inflammatory resolution research

This divergence underscores the need for subtype-specific targeting of autophagy

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