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Turbulent flow promotes NF-B and AP-1 activity that upregulate pro-inflammatory and pro-atherogenic genes such as ICAM-1, E-selectin, platelet-derived growth factor (PDGF)-BB, IL-1a, bone morphogenic protein-4 (BMP-4), monocyte chemotactic protein-1 (MCP-1), and ET-1 [211]

While senescence may initially serve as a protective mechanism, the long-term accumulation of senescent cells in tissues can contribute to various age-related pathological conditions

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Amylin is co-secreted with insulin from pancreatic beta cells

Pink1 and Parkin KO mice both showed an increase in increased inflammation, but antioxidants abolished CASP1 activation, suggesting a role for ROS in the inflammation associated with impaired mitophagy (Sliter et al., 2018)