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Conclusion: Here we report a novel function of portal fibroblasts as drivers of cholestatic fibrosis, ductular reaction, inflammation, hepatocyte senescence, and HCC in aged mice, demonstrating the key role of the tumor microenvironment

This enhanced structural integrity improves the skin's ability to shed damaged, hyperpigmented cells and replace them with healthy, evenly pigmented ones

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No tissue-level interaction is expected, but coordination prevents confusion in clinical records

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