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MSH3 deficiency specifically improves open-chromatin dysregulation in neurons

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Side effects can occur if the living drug multiplies too actively, the most common being cytokine release syndrome (CRS) and immune effector cell-associated neurotoxicity syndrome (ICANS)

Beyond that, GSK3 mediates Nrf2 degeneration, whereas AKT suppresses the activity of GSK3, which stabilizes Nrf2 to defend ferroptosis in IDH-mutated glioma[148]

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