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This suggests that under conditions of acute lung injury or pulmonary inflammation, the body may compensatorily increase N-acetyl-L-carnosinea precursor form that is less susceptible to enzymatic degradationto participate in a systemic anti-inflammatory response

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For, different overdose amount of APAP, we compare the NAPQI formation, the hepatocyte regeneration, glutathione production and APAP metabolism when alcohol is not ingested with APAP in the two models

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