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PMID: 30377225 Mol Cancer Ther Involvement of the FGF8/FGF receptor signaling pathway in the maintenance and progression of fusion-positive rhabdomyosarcoma

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During early disease, stimuli such as bleomycin (BLM) or lipopolysaccharide (LPS) induce ferroptosis in ATII cells, disrupting the alveolar barrier and intensifying oxidative stress ( 2 + in fibroblasts and promotes their myofibroblastic conversion, maintaining EMT-linked features ( Ferroptosis aggravates epithelial injury and, by generating ROS and activating pro-inflammatory pathways such as TGF- and NF-B, further promotes EMT, mesenchymal activation and tissue remodeling ( It is noteworthy that aberrant iron homeostasis forms the central hub of ferroptosis-EMT crosstalk

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