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Therefore, considering the presence of APAP protein adducts at the therapeutic dose of APAP, formation of protein adducts on mitochondrial proteins rather than the overall formation of protein adducts may be the reason for cellular toxicity, which has been considered a key factor for necrotic cell death in previous years (Jollow et al., 1973

(PDF 383 kb) Supplementary Data 1 (download XLSX Comparison of matrix concentrations of mitochondrial metabolites using normal and lengthened isolation times (.xls file)

This study showed that doses of 500-1000 mg were sufficient to reduce markers of oxidative stress

Circulating glutathione is primarily cleared by the kidney

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