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Experimental vitamin D deficiency is prevented from promoting the development of autoimmune disorders, such as inflammatory bowel disease, by the active form of vitamin D

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doi: 10.1002/ctm2.1564 50 WeiDSuYLeungPCKLiYChenZ-J

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Chang and colleagues (Journal of Applied Physiology, 2011) reported that BPC-157 promotes tendon outgrowth, cell survival, and migration in cultured tenocytes, findings that have been replicated by multiple independent groups and form the basis for its prominence in musculoskeletal-repair research