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doi: 10.4049/jimmunol.1501141 106 MourikBCLubbertsEde SteenwinkelJEMOttenhoffTHMLeenenPJM

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This work was supported by grants from National Institutes of Health and Walther Cancer Institute (K.L.G.)

4.2 Allergic asthma and allergic rhinitis An in vitro model of allergic airway inflammation, BEAS-2B human bronchial epithelial cells, were pre-treated with BBR (1 M) and activated, resulting in significant inhibition of IL-6 and CC motif chemokine ligand (CCL)-11 secretion, possibly through suppression of STAT6 signaling, without affecting cell viability [53]

"Estimation of net endogenous noncarbonicacid production in humans from diet potassium and protein contents." Am