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Interestingly, loss of its receptor SLC22A17 did not reverse this effect, suggesting that LCN2 modulates System Xc - -GPX4 pathway through mechanisms independent of SLC22A17

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Compared to the vehicle and GPE + GH groups, GPE effects in NSCs proliferation were shown by a significant increase in BrdU+ cells (representing labelled proliferating cells), while significant migration by GPEs in NSCs was presented by the wound closure rate, which was independent of cell proliferation