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Since the ECM of the TME contains abundant hyaluronidase, encapsulating nanodelivery carriers with an HA shell allows the nanocarriers to be degraded by hyaluronidase at the tumor site, resulting in accumulation at the tumor and increased drug delivery efficiency

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Serum irisin levels are decreased in patients with sepsis, and exogenous irisin suppresses ferroptosis in the liver of septic mice

These regulatory systems enable cancer cells to enhance their adaptive capacity when confronted with oxidative stress damage induced by treatment, thereby playing a pivotal role in the formation of DTP

The pathological consequences of chronic inflammation in cancer are profound: persistent cytokine signaling enhances tumor cell proliferation and survival, while upregulation of immune checkpoint molecules like PD-L1 suppresses cytotoxic T cell responses, facilitating immune evasion [48]