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Following transplantation, MGE cells migrated away from the injection site and spread across the host striatum

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however, in pathological conditions, the function of Na + /K + -ATPase (NKA) becomes dysfunctional, with subsequent disruption of Na + /K + electrochemical gradient needed for glutamate translocation, resulting in increases in extracellular K + and decreases in Na + decreases, which leads to glutamate transport in the outward direction [139]