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We recently discovered in a drug screen that Diphenyleneiodonium chloride (DPI) selectively induces cell death of MNA NB cells in vitro and decreases tumor growth in vivo ( 16)

Although its pathogenesis cannot be explained by a single hypothesis, several mechanisms are especially relevant to nanotherapeutic intervention, including neurotransmitter imbalance, hypothalamicpituitaryadrenal (HPA) axis dysregulation, neuroinflammation, oxidative stress, and impaired neuroplasticity (Fig

The balance between microbial arsenic methylation and demethylation in paddy soils underpins global arsenic risk and straighthead disease in rice