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Furthermore, the metabolites of BaP such as 7,8-dihydrodiol, 3,6-, and 6,12-dione metabolites are reported involvement in BaP induced abdominal aortic toxicity via elevating plasma ROS levels and increased protein expression of TNF, CYP1A1, and MMP9 ( BaP and myocardial injury In BaP-treated offspring rats, microarray and quantitative real-time PCR analysis revealed that the up-regulated gene expression of AngII, angiotensinogen and, eNOS, which are associated with the dysregulate cardiovascular development (76)

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