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This phenomenon may be related to the increased risk of macrovascular disease because of the upregulation of phenylalanine, which is the hydrolysis product of phenylacetylglycine, in plasma (67,68)

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BCMA expression and soluble BCMA levels have been correlated with diseases status and therapeutic response in MM, and additional BCMA-targeting modalities, including bispecific T-cell engagers and antibodydrug conjugates, are under active clinical investigation ( Although BCMA-targeted CAR T-cell therapies induce deep initial responses in many patients, disease relapses remain common, underscoring the urgent need for effective post-BCMA treatment strategies