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High glutamine impedes growth independent of BRAF status To further test the effect of increased dietary glutamine, we used two melanoma patient-derived xenograft (PDX) models with different genetic backgrounds, PDX_TM00702 (with wild-type BRAF) and PDX_TM01612 (with BRAF V600E mutation), and placed mice on either control or glutamine-rich diet when tumours reached an average of 50 mm 3

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This has been confirmed in mice with SQOR gene defects: loss of SQOR function leads to a fatal Leigh-like syndrome (Landry et al., 2021a

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Natural compounds typically have multiple pharmacological actions because they interact with various biological targets

Why This Matters for Patients and Clinicians The peptides on this agenda have developed substantial patient interest over the past several years