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These inhibitors, when administered either topically or orally, not only inhibited the cerebral vasodilatory responses but also inhibited or significantly decreased H 2 S elevations in the brain caused by both topical and oral SFN (101)

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Common observations include a reduction in certain beneficial genera (such as butyrate producers Roseburia and Faecalibacterium ) and an increase in pro-inflammatory genera (like Escherichia or Oscillospira in some reports) [213]