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It converts homocysteine into methionine, which helps protect the cardiovascular system

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While STARD1-dependent BA synthesis stimulates carcinogenesis through the induction of transcription factors involved in the stemness, self-renewal, and inflammation, the enrichment of mitochondria in cholesterol and subsequent impact on mitochondrial membrane fluidity impairs chemotherapy that targets the mitochondrial apoptotic pathway by restraining the mitochondrial outer membrane permeabilization and subsequent engagement of the apoptosome [135]