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found that hypoxia reduces NCOA4 expression in human macrophages, and siRNA transfection experiments showed that knocking down NCOA4 increases FtMt expression, protecting macrophages from RSL3-induced ferroptosis [20]

Once unfolded, it does not refold correctly

and ACSL4, ALOX12 and ALOX15, which are involved in lipid oxidation, are all known targets for driving ferroptosis

The loss of these mitochondrial protein activities leads to acceleration of oxidative and nitrosative stress