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However, due to the complexity of USP regulation, the precise mechanism of its interaction with ferroptosis is still unknown, which requires more future studies to confirm

These findings support the hypothesis that ferroptosis may be a critical participant in DILI, which is corroborated by recent investigations

We know that caloric restriction and other forms of fasting induce changes in the metabolism of these cells, forcing them to reduce their reliance on glucose and increase the use of mitochondrial oxidative pathways

In addition, we observed in histology that there was an increase in inflammatory cells, necrosis, hemorrhage and hyperemia

doi: 10.3892/mmr [DOI] [PMC free article] [PubMed] [Google Scholar] 64

This is why treatment should always be supervised by licensed professionals who can respond quickly