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To identify the exact location of binding sites for the predicted TFs, we detected the motif occurrences in the promoter regions ( 500 bp from a transcription start site) of genes from the gene sets using HOCOMOCO collection v.11 89

Based on these reported findings, we offer a model where reduced GCLC expression under relative glucose abundance potentially underlies PDAC sensitivity to chemotherapy ( Fig

380 Activated ERK1/2 promotes a pro-regenerative phenotype by releasing HGF and Wnt, while activated AKT induces a profibrotic phenotype

Significantly more or less abundant proteins were further subjected to GOBP using StringApp (v1.6.0) 79 and were additionally visualized using EnrichmentMap (v3.3.1) 80 , all in Cytoscape v3.9.1 81

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