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Indeed, its dysregulation would hinder the ability of cells to sustain metabolic needs and might contribute to the onset and development of some metabolic disorders including insulin resistance, diabetes mellitus, obesity, and atherosclerosis 8,9,10

When supporting similar pathways, urolithin A is often used in clinic

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(197) reported that curcumin inhibited oxidative stress and granulosa cell autophagy through activation of the Nrf2/HO-1 and PI3K/Akt signaling pathways, thereby improving ovarian morphology and function in D-gal-induced POF mice