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Clinically, these motor impairments such as bradykinesia, rigidity and tremors arise from the progressive loss of dopaminergic neurons in the substantia nigra pars compacta (SNpc) a pathological hallmark of PD [3]

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near research site (if localized SubQ modeling possible) or systemic administration for broader studies Cycle length: 46 weeks on for acute injury models (extend to 8 weeks if needed for chronic issues), followed by 24 weeks off to reset sensitivity Use strictly according to the manufacturers directions

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The primary antibodies were visualized using goat anti-rabbit Alexa Fluor 555 conjugate and goat anti-mouse Alexa Fluor 488 conjugate antibodies (1:800) (Cell Signaling Technology)