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Chiu CC, Keeling CI, Bohlmann J

Deletion of intestinal epithelial AMP-activated protein kinase alters distal colon permeability but not glucose homeostasis

May, 2019

Concurrently, increased aconitate serves as a substrate for aconitate decarboxylase 1 (ACOD1), producing itaconatean immunoregulatory metabolite that inhibits succinate dehydrogenase, stabilizes Nrf2 via KEAP1 alkylation, and suppresses excessive IL-1 and type I interferon responses and constrain inflammation 103,104

BA also regulates the Nrf2/Keap1 signalling pathway, restores HO-induced impaired nuclear maturation of oocytes, restores the levels of ROS and GSH, and acts as an effective antioxidant [126]

Infect Immun (2003) 71:49965004.10.1128/IAI.71.9.4996-5004.2003 72 Gruden-MovsesijanAIlicNMostarica-StojkovicMStosic-GrujicicSMilicMSofronic-MilosavljevicL