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Donorrecipient mismatch in FMT and poor colonization of many probiotic strains limit durable effect [274, 275, 321]

Genetic mechanisms of primary chemotherapy resistance in pediatric acute myeloid leukemia

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In addition, experimental studies have shown that the elevation of GSH can inhibit the activation of hepatic stellate cells [33], which is a key driving factor for extracellular matrix deposition and fibrosis, thereby preventing the progression of liver fibrosis [34]

Its main function is to neutralise free radicals and reduce oxidative stress, protecting cells from damage caused by environmental factors

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