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The localized mechanism explains the result: BPC-157 activates the fibroblasts that produce tendon collagen directly at the repair site

This potentially suggests that BPC-157 may activate the FAK-paxillin pathway, which may be involved in the promotion of tendon fibroblast migration and cell adhesion

Figure 1C), which subsequently results in the adaptor molecule binding with it and transmitting downstream signals (Sakai et al., 2015)

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