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Similarly, the same effect was observed in DLBCL cells with endogenous levels of GSH and the extent of GSH depletion was comparable to GSH depletion as a result of GSH synthesis inhibition using the -glutamylcysteine synthetase (-GCS) inhibitor buthionine sulphoximine (BSO) (Fig

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doi: 10.1080/00365521.2023.2173533 190 LakhanSEKirchgessnerA

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