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10.1016/S0002-9149(02)02250-6

To prevent proteotoxicity, the UPR not only increases protein-folding capacity but simultaneously reduces protein-folding load by stimulating proteasomal and lysosomal protein degradation

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Increased chondrocyte growth Improved collagen and proteoglycan expression (preclinical) Reduced inflammatory activity in joint-tissue models This is entirely lab-based, not clinical

In addition, the model of dry eye disease was established through inflammatory stimulation, and then trigeminal ganglion sensory neurons were isolated and co-cultured with corneal vascular endothelial cells

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