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Pandemic Mitigation CollaborativeCOVID-19 Forecasting Model

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Cell Immunol 130:5208, PMID: 2208308 Yamauchi A, Bloom ET (1997) Control of cell cycle progression in human natural killer cells through redox regulation of expression and phosphorylation of retinoblastoma gene product protein

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doi: 10.1093/hr/uhae043 10 ChandraS

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ALA effectively stabilizes hepatic redox-active iron (Fe 2+ ) expression and depresses the formation of ferroptosis after CSD Considering that overload of the redox-active iron (Fe 2+ ) plays a harmful role in hepatic metabolic function due to its involvement in catalyzing the Fenton reaction, generating ROS, enhancing oxidative stress, exacerbating inflammatory signaling, and leading to the development of ferroptosis, the present study further examined the hepatic Fe 2+ expression, together with the ferroptotic related proteins by the use of TOF-SIMS analysis and immunohistochemistry, respectively

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Olympus, Tokyo, Japan) and analyzed using ImageJ software (Java 1.8.0, National Institutes of Health, Bethesda, MD)

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Zhang, Dual Function of Magnesium in Bone Biomineralization

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