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Thirdly, through activation of nitric oxide synthase generating NO radicals which itself has been shown to disrupt Ca 2+ handling proteins (144, 145) but also to inhibit components of the respiratory chain and promote ROS production (146, 147)

Abdelraouf, K., Chang, K.-T., Yin, T., Hu, M

The heart is one of the organs most vulnerable to injury and dysfunction in sepsis, and sepsis-induced myocardial injury is relatively common in the ICU ( 2 S) ameliorates cardiac dysfunction in septic rats by reducing cardiomyocyte injury caused by excessive autophagy through the activation of the AMPK/mTOR pathway ( To assess the cardioprotective capacity of compound 3b-1 , we established an in vitro model of septic cardiomyopathy using lipopolysaccharide (LPS)-challenged H9c2 cardiomyocytes

WHO Expert Consultation

A Mendelian randomization analysis established causal links between specific GM and ALD, identifying two risk factors and one protective factor

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