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Description

& Burnett, A

Comorbid conditions, such as diabetes and chronic kidney disease, amplify ketamines neurotoxicity by increasing oxidative stress and impairing drug metabolism

MTX caused moderate tubular degeneration as well as loss of luminal border integrity of proximal and distal convoluted tubules, obvious congestion and dilatation of glomerular tuft capillaries, and other pathological changes

Data Availability Statement No new data were created or analyzed in this study

Through the glutamine-mediated "Trojan Horse" mechanism, ammonia accumulates in mitochondria, inducing mitochondrial permeability transition pore opening, mitochondrial swelling, and cytochrome c release, ultimately triggering mitochondrial dysfunction and apoptosis (Zielinska et al